The Dougherty Lab has a new preprint, “Gtf2i and Gtf2ird1 mutation are not sufficient to reproduce mouse phenotypes caused by the Williams Syndrome critical region.” In the paper, first author Nathan Kopp, graduate student in the Dougherty Lab, disprovesĀ a dominant hypothesis. Kopp and his fellow authors show “that the complete deletion model has deficits across several behavioral domains including social communication, motor functioning, and conditioned fear that are not explained by loss of function mutations in Gtf2i and Gtf2ird1,” they write in the paper. Congratulations, all!